Twelve years ago, a new form of cell death was discovered by a team of researchers at Columbia University. It’s called ferroptosis. Over 10,000 scientific papers have been published on ferroptosis, and we now know that it can accelerate aging, type 2 diabetes, heart disease, and fatty liver disease. The cause of ferroptosis has remained unknown. Until now.
As published in Metabolites, a new nutritional C15:0 (pentadecanoic acid) deficiency syndrome has been discovered. Cellular Fragility Syndrome is the first nutritional deficiency syndrome to be discovered in 75 years and may be affecting as many as 1 in 3 of us.
This paper is a culmination of 12 years of studies that explain C15:0’s role as an essential fatty acid, how low C15:0 levels results in cellular fragility, ferroptosis and impaired health, and most importantly, how replenishing our C15:0 levels fixes our cells, reverses ferroptosis, and restores metabolic, liver, and heart health.
Population wide declines in C15:0 levels have been occurring due to 1) lower intake of whole dairy-fat foods (our primary source of C15:0), 2) lower C15:0 content in dairy due to changing agricultural practices, and 3) aging – our C15:0 levels naturally decline as we get older.
The good news is that nutritional deficiency syndromes are detectable and fixable. It’s time to revisit nutritional guidelines and agriculture industry practices to get C15:0 back into our lives.
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